Why Do I Wake Up Right Before My Alarm Goes Off?

Because your body’s internal clock is just as good, if not better, than the contraption shrieking atop your nightstand.  

At the center of your brain, a clump of nerves—called the suprachiasmatic nucleus—oversees your body’s clock: the circadian rhythm. It determines when you feel sleepy and when you feel bright-eyed. It controls your blood pressure, your body temperature, and your sense of time. It turns your body into a finely tuned machine.

That machine happens to love predictability. Your body is most efficient when there’s a routine to follow. So if you hit the hay the same time each night and awake the same time each morning, your body locks that behavior in. And that’s where things get sciency.

Beat the clock!

Your sleep-wake cycle is regulated by a protein called PER. The protein level rises and falls each day, peaking in the evening and plummeting at night. When PER levels are low, your blood pressure drops, heart rate slows, and thinking becomes foggier. You get sleepy.

If you follow a diligent sleep routine—waking up the same time every day—your body learns to increase your PER levels in time for your alarm. About an hour before you’re supposed to wake up, PER levels rise (along with your body temperature and blood pressure). To prepare for the stress of waking, your body releases a cocktail of stress hormones, like cortisol. Gradually, your sleep becomes lighter and lighter. 

And that’s why you wake up before your alarm. Your body hates your alarm clock. It’s jarring. It’s stressful. And it ruins all that hard work. It defeats the purpose of gradually waking up. So, to avoid being interrupted, your body does something amazing: It starts increasing PER and stress hormones earlier in the night. Your body gets a head start so the waking process isn’t cut short. It’s so precise that your eyelids open minutes—maybe even seconds—before the alarm goes off.

You snooze, you lose

There’s evidence you can will yourself to wake on time, too. Sleep scientists at Germany’s University of Lubeck asked 15 volunteers to sleep in their lab for three nights. One night, the group was told they’d be woken at 6 a.m., while on other nights the group was told they’d be woken at 9 a.m..

But the researchers lied—they woke the volunteers at 6 a.m anyway. And the results were startling. The days when sleepers were told they’d wake up early, their stress hormones increased at 4:30 a.m., as if they were anticipating an early morning. When the sleepers were told they’d wake up at 9 a.m., their stress hormones didn’t increase—and they woke up groggier. “Our bodies, in other words, note the time we hope to begin our day and gradually prepare us for consciousness,” writes Jeff Howe at Psychology Today.

Incidentally, if you don’t wake before your alarm, you probably aren’t getting enough sleep—or you aren’t sleeping on a consistent schedule. Waking up at different times on weekdays and weekends can quickly throw your clock out of whack. Without any consistency, your body may not know when to get up. So when your alarm starts screaming, you feel dazed and grumpy.

Enter the snooze button. Since your body’s gone through all that work to rise gradually, a quick nap sends your internal clock spinning in the wrong direction. All the hormones that help you fall asleep meddle with the hormones that help you wake up. Your body gets confused. You feel groggier. And with each slap of the snooze, it gets worse. The snooze, it seems, is the worst way to start your day.

What Do the Numbers and Letters on a Boarding Pass Mean?

iStock.com/Laurence Dutton
iStock.com/Laurence Dutton

Picture this: You're about to embark on a vacation or business trip, and you have to fly to reach your destination. You get to the airport, make it through the security checkpoint, and breathe a sigh of relief. What do you do next? After putting your shoes back on, you'll probably look at your boarding pass to double-check your gate number and boarding time. You might scan the information screen for your flight number to see if your plane will arrive on schedule, and at some point before boarding, you'll also probably check your zone and seat numbers.

Aside from these key nuggets of information, the other letters and numbers on your boarding pass might seem like gobbledygook. If you find this layout confusing, you're not the only one. Designer and creative director Tyler Thompson once commented that it was almost as if "someone put on a blindfold, drank a fifth of whiskey, spun around 100 times, got kicked in the face by a mule … and then just started puking numbers and letters onto the boarding pass at random."

Of course, these seemingly secret codes aren't exactly secret, and they aren't random either. So let's break it down, starting with the six-character code you'll see somewhere on your boarding pass. This is your Passenger Name Reference (or PNR for short). On some boarding passes—like the one shown below—it may be referred to as a record locator or reservation code.

A boarding pass
Piergiuliano Chesi, Wikimedia Commons // Public domain

These alphanumeric codes are randomly generated, but they're also unique to your personal travel itinerary. They give airlines access to key information about your contact information and reservation—even your meal preferences. This is why it's ill-advised to post a photo of your boarding pass to social media while waiting at your airport gate. A hacker could theoretically use that PNR to access your account, and from there they could claim your frequent flier miles, change your flight details, or cancel your trip altogether.

You might also see a random standalone letter on your boarding pass. This references your booking class. "A" and "F," for instance, are typically used for first-class seats. The letter "Y" generally stands for economy class, while "Q" is an economy ticket purchased at a discounted rate. If you see a "B" you might be in luck—it means you could be eligible for a seat upgrade.

There might be other letters, too. "S/O," which is short for stopover, means you have a layover that lasts longer than four hours in the U.S. or more than 24 hours in another country. Likewise, "STPC" means "stopover paid by carrier," so you'll likely be put up in a hotel free of charge. Score!

One code you probably don’t want to see is "SSSS," which means your chances of getting stopped by TSA agents for a "Secondary Security Screening Selection" are high. For whatever reason, you've been identified as a higher security risk. This could be because you've booked last-minute or international one-way flights, or perhaps you've traveled to a "high-risk country." It could also be completely random.

Still confused? For a visual of what that all these codes look like on a boarding pass, check out this helpful infographic published by Lifehacker.

Have you got a Big Question you'd like us to answer? If so, send it to bigquestions@mentalfloss.com.

Does Having Allergies Mean That You Have A Decreased Immunity?


Tirumalai Kamala:

No, allergy isn't a sign of decreased immunity. It is a specific type of immune dysregulation. Autoimmunity, inflammatory disorders such as IBS and IBD, and even cancer are examples of other types of immune dysregulation.

Quality and target of immune responses and not their strength is the core issue in allergy. Let's see how.

—Allergens—substances known to induce allergy—are common. Some such as house dust mite and pollen are even ubiquitous.
—Everyone is exposed to allergens yet only a relative handful are clinically diagnosed with allergy.
—Thus allergens don't inherently trigger allergy. They can but only in those predisposed to allergy, not in everyone.
—Each allergic person makes pathological immune responses to not all but to only one or a few structurally related allergens while the non-allergic don't.
—Those diagnosed with allergy aren't necessarily more susceptible to other diseases.

If the immune response of each allergic person is selectively distorted when responding to specific allergens, what makes someone allergic? Obviously a mix of genetic and environmental factors.

[The] thing is allergy prevalence has spiked in recent decades, especially in developed countries, [which is] too short a time period for purely genetic mutation-based changes to be the sole cause, since that would take multiple generations to have such a population-wide effect. That tilts the balance towards environmental change, but what specifically?

Starting in the 1960s, epidemiologists began reporting a link between infections and allergy—[the] more infections in childhood, [the] less the allergy risk [this is called hygiene hypothesis]. Back then, microbiota weren't even a consideration but now we have learned better, so the hygiene hypothesis has expanded to include them.

Essentially, the idea is that the current Western style of living that rapidly developed over the 20th century fundamentally and dramatically reduced lifetime, and, crucially, early life exposure to environmental microorganisms, many of which would have normally become part of an individual's gut microbiota after they were born.

How could gut microbiota composition changes lead to selective allergies in specific individuals? Genetic predisposition should be taken as a given. However, natural history suggests that such predisposition transitioned to a full fledged clinical condition much more rarely in times past.

Let's briefly consider how that equation might have fundamentally changed in recent times. Consider indoor sanitation, piped chlorinated water, C-sections, milk formula, ultra-processed foods, lack of regular contact with farm animals (as a surrogate for nature) and profligate, ubiquitous, even excessive use of antimicrobial products such as antibiotics, to name just a few important factors.

Though some of these were beneficial in their own way, epidemiological data now suggests that such innovations in living conditions also disrupted the intimate association with the natural world that had been the norm for human societies since time immemorial. In the process such dramatic changes appear to have profoundly reduced human gut microbiota diversity among many, mostly in developed countries.

Unbeknownst to us, an epidemic of absence*, as Moises Velasquez-Manoff evocatively puts it, has thus been invisibly taking place across many human societies over the 20th century in lock-step with specific changes in living standards.

Such sudden and profound reduction in gut microbiota diversity thus emerges as the trigger that flips the normally hidden predisposition in some into clinically overt allergy. Actual mechanics of the process remain the subject of active research.

We (my colleague and I) propose a novel predictive mechanism for how disruption of regulatory T cell** function serves as the decisive and non-negotiable link between loss of specific microbiota and inflammatory disorders such as allergies. Time (and supporting data) will tell if we are right.

* An Epidemic of Absence: A New Way of Understanding Allergies and Autoimmune Diseases Reprint, Moises Velasquez-Manoff

** a small indispensable subset of CD4+ T cells.

This post originally appeared on Quora. Click here to view.