8 Myths About Dead Bodies You Probably Think Are True

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iStock

Bodies are weird enough, but it's the dead ones that hold real intrigue. The fact that most of us just don't spend that much time around them means it's hard to separate truth from fiction; corpses have been thought to be responsible for plagues, as well as to carry magic healing properties. Below, some dead body myths that won't give up the ghost—and explanations for the real-life science behind them.

1. HAIR AND NAILS GROW AFTER DEATH.

Corpse under sheet with hand sticking out

Not true! The cell division driving hair and nail growth stops when the body dies and the heart no longer pumps oxygen-filled blood throughout the circulatory system. It does look like things keep growing, though. When a dead body's skin loses hydration, it retracts—and retraction along the nail bed makes it appear as if the nails are getting longer. As for hair, drying skin on the face and head "pulls back towards the skull, making stubble appear more prominent," writes Claudia Hammond for the BBC. "Goosebumps caused by the contraction of the hair muscles can add to the effect."

2. DEAD BODIES ARE DANGEROUS.

There's no science to back up the idea that a dead and decomposing body is harmful to the living just by virtue of its being dead. This might sound obvious, but the belief that disease came from breathing in air infected by corpses was once common.

Miasmatic theory, as it was called, was a widespread belief among members of the medical profession (and the public) in the 19th century. Miasma, an ancient Greek word for "pollution," was the bad air coming from "rotting corpses, the exhalations of other people already infected, sewage, or even rotting vegetation" and was thought to be responsible for the spread of disease. Fortunately, this belief was eventually replaced by germ theory.

3. … AND MULTIPLE DEAD BODIES ARE EXTRA DANGEROUS.

In a publication from the Pan American Health Organization (a division of the World Health Organization), Donna Eberwine explains that the belief that dead bodies spread disease "remains a chronic problem in disaster relief efforts." After natural disasters, there is often a hysteria around dead bodies and a rush to immediately bury them, which distracts relief efforts from more pressing concerns. "The microorganisms that are involved in decomposition are not the kind that cause disease," Eberwine writes. "And most viruses and bacteria that do cause disease cannot survive more than a few hours in a dead body."

There are some exceptions. The level of Ebola virus in dead victims remains high, and their remains should only be handled by people in protective gear (and buried quickly). HIV can live for up to 16 days in a body held under refrigeration, and other blood-borne viruses like hepatitis, along with tuberculosis and gastrointestinal infections, can pose a risk. "The risk of contagion can be minimized with basic precautions and proper hygiene," Eberwine writes.

4. EMBALMING MAKES DEAD BODIES "SAFER."

Egyptian sarcophagus

"Embalming provides no public health benefit," according to the Funeral Consumer's Alliance (a nonprofit focused on affordable death care), citing the Centers for Disease Control and Canadian authorities. While individual morticians might say that a body must be embalmed before viewing, burial, or cremation, the process is generally not legally required. Moreover, since a dead body is usually not in itself harmful, embalming does not make it any safer. On the flip side, embalming chemicals are actually quite toxic, and embalmers must cover their entire body and wear a respirator while working. 

5. DEAD BODIES SIT UP ON THE MEDICAL TABLE.

This horror-movie trope just isn't real. During decomposition, a body might twitch or make small movements and noises due to the gas and waste released by bacteria. A decomposing corpse can definitely move a little, but sitting straight up is just not going to happen.

6. BURYING A BODY WITHOUT A COFFIN OR VAULT MEANS IT WILL CONTAMINATE THE GROUNDWATER.

Nope! Burials usually occur at 3.5 feet below the surface, whereas water can be 75 feet underground. "Mandatory setbacks from known water sources also ensure that surface water is not at risk," the Green Burial Council explains [PDF]. Additionally, because microorganisms living in the soil will break down the chemical compounds that remain in a dead body, we actually give out "more toxic chemicals during a day of living than a whole body will decomposing."

7. CREMAINS ARE "ASH."

Wall of cremation urns

Though we often talk of "scattering ashes," cremains are a little more complicated. Once a body intended for cremation has been burned in what's called a retort, what's left will be put in a cremulator. Sort of like a blender, the cremulator uses ball bearings or rotating blades to pulverize the bones and other remnants into a "grayish, coarse material, like fine gravel," as HowStuffWorks puts it.

8. ALL IN ALL, MAYBE DEATH ISN'T AS SCARY AS WE THINK.

According to psychological scientist Kurt Gray, it's possible that death isn't quite as terrifying as we think it is. Gray studied the responses of death row inmates and terminally ill patients as well as those of people asked to imagine they had untreatable cancer, and found that "while it's natural to fear death in the abstract, the closer one actually gets to it, the more positive he or she becomes," as New York Magazine explains. This may be due to something called the "psychological immune system," a term coined by Harvard psychologist Dan Gilbert in his book Stumbling on Happiness. According to Gray, our psychological immune system is engaged when bad things happen. "So when one is faced with death, all sorts of rationalization and meaning-making processes come in," he told New York Magazine. That may sound like your brain's trying to give you a cop-out, but it's much better than living in terror.

All photos courtesy of iStock.

12 Facts About Diabetes Mellitus

iStock/mthipsorn
iStock/mthipsorn

Thirty million Americans—about 9 percent of the country's population—are living with diabetes mellitus, or simply diabetes. This chronic condition is characterized by sustained high blood sugar levels. In many patients, symptoms can be managed with insulin injections and lifestyle changes, but in others, the complications can be deadly. Here's what you need to know about diabetes mellitus.

1. There are three types of diabetes.

In healthy people, the pancreas produces enough of the hormone insulin to metabolize sugars into glucose and move the glucose into cells, where it's used for energy.

But people with type 2 diabetes—the most common form of the disease, accounting for about 95 percent of cases—either can't produce enough insulin to transport the sugars, or their cells have become insulin-resistant. The result is a buildup of glucose in the blood (a.k.a. high blood sugar or hyperglycemia). Type 2 diabetes typically develops in adults.

Type 1 diabetes, also known as juvenile diabetes, makes up the remaining 5 percent of chronic cases and most often develops in children and young adults. With this condition, the initial problem isn’t blood sugar levels, but insulin production: The pancreas can’t make enough insulin to process even normal amounts of glucose. The sugar builds up as a result, leading to dangerous concentrations in the bloodstream.

The third form, gestational diabetes, only afflicts pregnant people who weren’t diabetic before their pregnancy. The mother's blood glucose levels usually spike around the 24th week of pregnancy, but with a healthy diet, exercise, and insulin shots in some cases, diabetes symptoms usually can be managed. Blood sugar levels tend to return to normal in patients following their pregnancies.

2. The mellitus in diabetes mellitus means "honey sweet."

Around 3000 years ago, ancient Egyptians described a condition with diabetes-like symptoms, though it wasn't called diabetes yet. It took a few hundred years before the Greek physician Araetus of Cappodocia came up with the name diabetes based on the Greek word for "passing through" (as in passing a lot of urine, a common diabetes symptom). English doctor Thomas Willis tacked on the word mellitus, meaning "honey sweet," in 1675, building on previous physicians' observations that diabetic patients had sweet urine. Finally, in 1776, another English physician named Matthew Dobson confirmed that both the blood and urine of diabetes patients were made sweeter by high levels of glucose in their blood.

3. The cause of one type of diabetes is well understood; the other, not so much.

A person’s lifestyle is a key predictor of developing type 2 diabetes. Factors like being overweight or obese, consuming a high-calorie diet, smoking, and seldom exercising contribute to the risk. Foods and drinks that are high in sugar—soda, candy, ice cream, dessert— may contribute to hyperglycemia, but any food that’s high in calories, even if it's not sweet, can raise blood sugar levels.

In contrast to these well-established factors, medical experts aren’t entirely sure what causes type 1 diabetes. We do know that type 1 is an autoimmune disease that develops when the body attacks and damages insulin-producing cells in the pancreas. Some scientists think that environmental factors, like viruses, may trigger this immune response.

4. Family history also plays a role in diabetes risk.

If a parent or sibling has type 2 diabetes, you are predisposed to developing pre-diabetes and type 2 diabetes. Lifestyle habits explain some of these incidences, since family members may share similar diets and exercise habits. Genetics also play a role, but just because one close relative has diabetes does not mean you're destined to. Research conducted on identical twins, which share identical genes, showed that the pairs have discordant risk. Among twins in which one has type 1 diabetes, the other has only a 50 percent chance of developing it; for type 2, the risk for the second twin is 75 percent at most.

5. Racial minorities are at a higher risk for developing diabetes.

Many racial minority groups in the U.S. have a higher chance of developing type 2 diabetes. Black Americans, Latino Americans, Native Americans, Pacific Islanders, and some groups of Asian Americans are more likely to have pre-diabetes and type 2 diabetes than white Americans. This can be partly explained by the fact that some of these groups also have higher rates of obesity, which is one of the primary risk factors of type 2 diabetes. Socioeconomics may also play a role: One study shows that people with diabetes living in poverty are less likely to visit diabetes clinics and receive proper testing than their middle-income counterparts. According to another study, diabetic people without health insurance have higher blood sugar, blood pressure, and cholesterol rates than insured diabetics. Genetics, on the other hand, don’t appear to contribute to these trends.

6. Diabetes is one of the world's deadliest diseases.

With proper management, people with diabetes can live long, comfortable lives. But if the disease isn’t treated, it can have dire consequences. Diabetics make up the majority of people who develop chronic kidney disease, have adult-onset blindness, and need lower-limb amputations. In the most serious cases, diabetes leads to death. The condition is one of the deadliest diseases in the world, killing more people than breast cancer and AIDS combined.

7. Millions of Americans are pre-diabetic.

According to the CDC, 84 million adults living in the U.S. are pre-diabetic: Their blood sugar is higher than what’s considered safe, but hasn't yet reached diabetic level. In pre-diabetic patients, blood glucose levels after eight hours of fasting fall between 100 and 125 milligrams per deciliter, and diabetic levels are anything above that. People with pre-diabetes are not just at a greater risk for type 2 diabetes, but also for heart disease and stroke. Fortunately, people who are diagnosed with pre-diabetes can take steps to eat a healthier diet, increase physical activity, and test their blood glucose level several times a day to control the condition. In some cases, doctors will prescribe drugs like metformin that make the body more receptive to the insulin it produces.

8. After climbing for decades, rates of diabetes incidence are declining.

In the U.S., the rate of new diagnoses skyrocketed 382 percent between 1988 and 2014. Globally, 108 million people had diabetes in 1980, but by 2014 that number was 422 million.

But thanks to nationwide education and prevention efforts, the trend has reversed in the U.S., according to the CDC. Since peaking in 2009, the number of new diabetes cases in America has dropped by 35 percent. In that same timeframe, the number of people living with diagnosed diabetes in the U.S. has plateaued, suggesting people with the condition are living longer.

9. The first successful treatment for type 1 diabetes occurred in 1922.

Prior to the 20th century, type 1 diabetes was usually fatal. Diabetic ketoacidosis—a toxic buildup of chemicals called ketones, which arise when the body can no longer use glucose and instead breaks down other tissues for energy—killed most patients within a year or two of diagnosis. In searching for way to save children with juvenile (type 1) diabetes, Canadian physician Frederick Banting and medical student Charles Best built on the work of earlier researchers, who had demonstrated that removing the pancreas from a dog immediately caused diabetes symptoms in the animal. Banting and Best extracted insulin from dog pancreases in University of Toronto professor J.J.R. Macleod's lab. After injecting the insulin back into dogs whose pancreases had been removed, they realized the hormone regulated blood sugar levels. On January 11, 1922, they administered insulin to a human patient, and further refined the extract to reduce side effects. In 1923, Banting and Macleod received the Nobel Prize in Medicine for their work.

10. A pioneering physicist discovered the difference between type and and type 1 diabetes.

In the 1950s, physicist Rosalyn Yalow and her research partner Solomon Berson developed a method for measuring minute amounts of substances in blood. Inspired by Yalow's husband's struggle with diabetes, Yalow focused her research on insulin. Their "radioimmunoassay" technology revealed that some diabetes patients were still able to produce their own insulin, leading them to create two separate categories for the disease: “insulin-dependent” (type 1) and “non-insulin-dependent” (type 2). Prior to that discovery in 1959, there was no distinction between the two types. In 1977, Yalow won the 1977 Nobel Prize in Medicine for the radioimmunoassay, one of only 12 female Nobel laureates in medicine.

11. Making one insulin dose once required tons of pig parts.

Insulin is relatively easy to make today. Most of what's used in injections comes from a special non-disease-producing laboratory strain of E. coli bacteria that's been genetically modified to produce insulin, but that wasn't always the case. Until about 40 years ago, 2 tons of pig pancreases were required to produce just 8 ounces of pure insulin. The pig parts were typically recycled from pork farms.

12. A quarter of diabetes patients don’t know they have it.

The symptoms of type 2 diabetes can develop for years before patients think to ask their doctor about them. These include frequent urination, unexplained thirst, numbness in the extremities, dry skin, blurry vision, fatigue, and sores that are slow to heal—signs that may not be a cause for concern on their own, but together can indicate a more serious problem. Patients with type 1 diabetes may also experience nausea, vomiting, and stomach pain.

While serious, the symptoms of diabetes are sometimes easy to overlook. That’s why 25 percent of people with the illness, 7.2 million in the U.S., are undiagnosed. And that number doesn’t even cover the majority of people with pre-diabetes who aren’t aware they’re on their way to becoming diabetic.

There Are 2373 Squirrels in New York's Central Park, Census Finds

iStock/maximkabb
iStock/maximkabb

Central Park in New York City is home to starlings, raccoons, and exotic zoo animals, but perhaps the most visible fauna in the area are the eastern gray squirrels. Thanks to a team of citizen scientists, we now know exactly how many of the rodents occupy the space—approximately 2373 of them, according to a census reported by Smithsonian.

In October 2018, a group called the Squirrel Census—with help from the Explorers Club, the NYU Department of Environmental Studies, Macaulay Honors College, the Central Park Conservancy, and the New York City Department of Parks & Recreation—organized a squirrel survey across all 840 acres of Central Park. For 11 days, more than 300 volunteers staked out their sections of the park twice a day—at dawn and dusk when the crepuscular animals are most active—and noted each squirrel they spotted. They also recorded how the squirrels looked, vocalized, behaved, and reacted to humans.

The research was analyzed and presented at an Explorers Club event in New York City on June 20. All the non-peer-reviewed findings—which includes a printed report, an audio report on a vinyl 45, 37 pages of data, collectible squirrel cards, and large maps of the park and the squirrel locations—are available to purchase for $75 from the Squirrel Census website.

This isn't the first time a massive census has been conducted of a public park's squirrel population. In 2011, the Squirrel Census launched with its first survey of Atlanta's Inman Park. They've conducted satellite squirrel counts at other parks, but Central Park is just the second park the organization has investigated in person.

[h/t Smithsonian]

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