How Did the Duck Hunt Gun Work?

For many children of the '80s, a good portion of your childhood probably revolved around sitting too close to the TV, clutching a plastic safety cone-colored hand gun and blasting waterfowl out of a pixilated sky in Duck Hunt (also, trying to blow that dog’s head off when he laughed at you). The Duck Hunt gun, officially called the Nintendo Entertainment System (NES) Zapper, seems downright primitive next to the Nintendo’s Wii and Microsoft’s Kinect, but in the late 80s, it filled plenty of young heads with wonder. How did that thing work?

Annie get your Zapper

The Zapper’s ancestry goes back to the mid 1930s, when the first so-called “light guns” appeared after the development of light-sensing vacuum tubes. In the first light gun game, Ray-O-Lite (developed in 1936 by Seeburg, a company that made parts and systems for jukeboxes), players shot at small moving targets mounted with light sensors using a gun that emitted a beam of light. When the beam struck a sensor, the targets – ducks, coincidentally – registered the “hit” and a point was scored.

Light guns hit home video game consoles with Shooting Gallery on the Magnavox Odyssey in 1972. Because the included shotgun-style light gun was only usable on a Magnavox television, the game flopped. The Nintendo Entertainment System (NES) Zapper then fell into the hands of American kids in October 1985, when it was released in a bundle with the NES, a controller and a few games. Early versions of the peripheral were dark gray, but the color of the sci-fi ray gun-inspired Zapper was changed a few years later when a federal regulation required that toy and imitation firearms be “blaze orange” (color #12199, to be exact) so they wouldn’t be mistaken for the real deal.

While there were a number of Zapper-compatible games released for the NES (when I was a kid and my dad worked from home, we wasted plenty of afternoons away playing Hogan’s Alley), most lived in the shadow of the iconic Duck Hunt, the most recognizable and popular Zapper game.

Gone in a Flash

While older light guns like the Ray-O-Lite rifle emitted beams of light, the Zapper and many other recent light guns work by receiving light through a photodiode on or in the barrel and using that light to figure out where on the TV screen you're aiming.

When you point at a duck and pull the trigger, the computer in the NES blacks out the screen and the Zapper diode begins reception. Then, the computer flashes a solid white block around the targets you’re supposed to be shooting at. The photodiode in the Zapper detects the change in light intensity and tells the computer that it’s pointed at a lit target block — in others words, you should get a point because you hit a target. In the event of multiple targets, a white block is drawn around each potential target one at a time. The diode’s reception of light combined with the sequence of the drawing of the targets lets the computer know that you hit a target and which one it was. Of course, when you’re playing the game, you don’t notice the blackout and the targets flashing because it all happens in a fraction of a second.

This target flashing method helped Nintendo overcome a weakness of older light gun games: cheaters racking up high scores by pointing the gun at a steady light source, like a lamp, and hitting the first target right out of the gate.

If you’re hungry for a more technical depth, check out Nintendo's 1989 patent on the Zapper technology

What Do the Numbers and Letters on a Boarding Pass Mean? Dutton Dutton

Picture this: You're about to embark on a vacation or business trip, and you have to fly to reach your destination. You get to the airport, make it through the security checkpoint, and breathe a sigh of relief. What do you do next? After putting your shoes back on, you'll probably look at your boarding pass to double-check your gate number and boarding time. You might scan the information screen for your flight number to see if your plane will arrive on schedule, and at some point before boarding, you'll also probably check your zone and seat numbers.

Aside from these key nuggets of information, the other letters and numbers on your boarding pass might seem like gobbledygook. If you find this layout confusing, you're not the only one. Designer and creative director Tyler Thompson once commented that it was almost as if "someone put on a blindfold, drank a fifth of whiskey, spun around 100 times, got kicked in the face by a mule … and then just started puking numbers and letters onto the boarding pass at random."

Of course, these seemingly secret codes aren't exactly secret, and they aren't random either. So let's break it down, starting with the six-character code you'll see somewhere on your boarding pass. This is your Passenger Name Reference (or PNR for short). On some boarding passes—like the one shown below—it may be referred to as a record locator or reservation code.

A boarding pass
Piergiuliano Chesi, Wikimedia Commons // Public domain

These alphanumeric codes are randomly generated, but they're also unique to your personal travel itinerary. They give airlines access to key information about your contact information and reservation—even your meal preferences. This is why it's ill-advised to post a photo of your boarding pass to social media while waiting at your airport gate. A hacker could theoretically use that PNR to access your account, and from there they could claim your frequent flier miles, change your flight details, or cancel your trip altogether.

You might also see a random standalone letter on your boarding pass. This references your booking class. "A" and "F," for instance, are typically used for first-class seats. The letter "Y" generally stands for economy class, while "Q" is an economy ticket purchased at a discounted rate. If you see a "B" you might be in luck—it means you could be eligible for a seat upgrade.

There might be other letters, too. "S/O," which is short for stopover, means you have a layover that lasts longer than four hours in the U.S. or more than 24 hours in another country. Likewise, "STPC" means "stopover paid by carrier," so you'll likely be put up in a hotel free of charge. Score!

One code you probably don’t want to see is "SSSS," which means your chances of getting stopped by TSA agents for a "Secondary Security Screening Selection" are high. For whatever reason, you've been identified as a higher security risk. This could be because you've booked last-minute or international one-way flights, or perhaps you've traveled to a "high-risk country." It could also be completely random.

Still confused? For a visual of what that all these codes look like on a boarding pass, check out this helpful infographic published by Lifehacker.

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Does Having Allergies Mean That You Have A Decreased Immunity?

Tirumalai Kamala:

No, allergy isn't a sign of decreased immunity. It is a specific type of immune dysregulation. Autoimmunity, inflammatory disorders such as IBS and IBD, and even cancer are examples of other types of immune dysregulation.

Quality and target of immune responses and not their strength is the core issue in allergy. Let's see how.

—Allergens—substances known to induce allergy—are common. Some such as house dust mite and pollen are even ubiquitous.
—Everyone is exposed to allergens yet only a relative handful are clinically diagnosed with allergy.
—Thus allergens don't inherently trigger allergy. They can but only in those predisposed to allergy, not in everyone.
—Each allergic person makes pathological immune responses to not all but to only one or a few structurally related allergens while the non-allergic don't.
—Those diagnosed with allergy aren't necessarily more susceptible to other diseases.

If the immune response of each allergic person is selectively distorted when responding to specific allergens, what makes someone allergic? Obviously a mix of genetic and environmental factors.

[The] thing is allergy prevalence has spiked in recent decades, especially in developed countries, [which is] too short a time period for purely genetic mutation-based changes to be the sole cause, since that would take multiple generations to have such a population-wide effect. That tilts the balance towards environmental change, but what specifically?

Starting in the 1960s, epidemiologists began reporting a link between infections and allergy—[the] more infections in childhood, [the] less the allergy risk [this is called hygiene hypothesis]. Back then, microbiota weren't even a consideration but now we have learned better, so the hygiene hypothesis has expanded to include them.

Essentially, the idea is that the current Western style of living that rapidly developed over the 20th century fundamentally and dramatically reduced lifetime, and, crucially, early life exposure to environmental microorganisms, many of which would have normally become part of an individual's gut microbiota after they were born.

How could gut microbiota composition changes lead to selective allergies in specific individuals? Genetic predisposition should be taken as a given. However, natural history suggests that such predisposition transitioned to a full fledged clinical condition much more rarely in times past.

Let's briefly consider how that equation might have fundamentally changed in recent times. Consider indoor sanitation, piped chlorinated water, C-sections, milk formula, ultra-processed foods, lack of regular contact with farm animals (as a surrogate for nature) and profligate, ubiquitous, even excessive use of antimicrobial products such as antibiotics, to name just a few important factors.

Though some of these were beneficial in their own way, epidemiological data now suggests that such innovations in living conditions also disrupted the intimate association with the natural world that had been the norm for human societies since time immemorial. In the process such dramatic changes appear to have profoundly reduced human gut microbiota diversity among many, mostly in developed countries.

Unbeknownst to us, an epidemic of absence*, as Moises Velasquez-Manoff evocatively puts it, has thus been invisibly taking place across many human societies over the 20th century in lock-step with specific changes in living standards.

Such sudden and profound reduction in gut microbiota diversity thus emerges as the trigger that flips the normally hidden predisposition in some into clinically overt allergy. Actual mechanics of the process remain the subject of active research.

We (my colleague and I) propose a novel predictive mechanism for how disruption of regulatory T cell** function serves as the decisive and non-negotiable link between loss of specific microbiota and inflammatory disorders such as allergies. Time (and supporting data) will tell if we are right.

* An Epidemic of Absence: A New Way of Understanding Allergies and Autoimmune Diseases Reprint, Moises Velasquez-Manoff

** a small indispensable subset of CD4+ T cells.

This post originally appeared on Quora. Click here to view.